Propranolol may increase oxygen requirements by increasing left ventricular fiber length, end diastolic pressure, and systolic ejection period. Possible deleterious effects from long-term use of Propranolol have not yet been adequately appraised. Propranolol is a competitive antagonist which specifically competes with beta-adrenergic receptor stimulating agents for available beta-receptor sites. The oxygen requirement of the heart and so decreases its demand for oxygen. Several reports in the published literature describe cases in which Propranolol was used as a suicide agent. Propranolol should be administered with caution since it may block bronchodilation produced by endogenous and exogenous catecholamine stimulation of beta-adrenergic receptors. If you have ischemic heart disease. You should not stop taking this medicine suddenly, particularly if you have ischemic heart disease. Propranolol may increase oxygen requirements by increasing left ventricular fiber length, end diastolic pressure, and the velocity and extent of myocardial contraction.
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